Collaborations and publications

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* Pruijm, M., et al., Associations of Urinary Uromodulin with Clinical Characteristics and Markers of Tubular Function in the General Population. Clin J Am Soc Nephrol, 2015. [http://www.ncbi.nlm.nih.gov/pubmed/26683888]
 
* Pruijm, M., et al., Associations of Urinary Uromodulin with Clinical Characteristics and Markers of Tubular Function in the General Population. Clin J Am Soc Nephrol, 2015. [http://www.ncbi.nlm.nih.gov/pubmed/26683888]
  
<ins>Under current revision in Psychoneuroendocrinology (22.12.2015)</ins><br/>
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<ins>''Under current revision in Psychoneuroendocrinology (22.12.2015)''</ins>
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* Petrovic, D., et al. Sociodemographic, behavioral and genetic determinants of allostatic load in a Swiss population-based study
 
* Petrovic, D., et al. Sociodemographic, behavioral and genetic determinants of allostatic load in a Swiss population-based study
  

Revision as of 10:56, 22 December 2015

2015

  • Ackermann, D., et al., CYP17A1 Enzyme Activity Is Linked to Ambulatory Blood Pressure in a Family-Based Population Study. Am J Hypertens, 2015. [1]
  • Alwan, H., et al., Heritability of ambulatory and office blood pressure in the Swiss population. J Hypertens, 2015. 33(10): p. 2061-7. [2]
  • Canivell, S., et al., 4B.05: PLASMA COPEPTIN IS ASSOCIATED WITH INSULIN RESISTANCE IN A SWISS POPULATION-BASED STUDY. J Hypertens, 2015. 33 Suppl 1: p. e54. [3]
  • Forni Ogna, V., et al., New anthropometry-based age- and sex-specific reference values for urinary 24-hour creatinine excretion based on the adult Swiss population. BMC Med, 2015. 13: p. 40. [4]
  • Guessous, I., et al., Associations of ambulatory blood pressure with urinary caffeine and caffeine metabolite excretions. Hypertension, 2015. 65(3): p. 691-6. [5]
  • Guessous, I., et al., 1C.06: AMBULATORY PULSE PRESSURE IS NEGATIVELY ASSOCIATED WITH EXCRETIONS OF URINARY CAFFEINE AND ITS METABOLITES. J Hypertens, 2015. 33 Suppl 1: p. e10-1. [6]
  • Moulin, F., et al., 5C.09: HERITABILITY OF RENAL FUNCTION PARAMETERS AND ELECTROLYTE LEVELS IN THE SWISS POPULATION. J Hypertens, 2015. 33 Suppl 1: p. e70. [7]
  • Pivin, E., et al., Inactive Matrix Gla-Protein Is Associated With Arterial Stiffness in an Adult Population-Based Study. Hypertension, 2015. 66(1): p. 85-92. [8]
  • Pivin, E., et al., 1D.03: INACTIVE MATRIX GLA PROTEIN IS ASSOCIATED WITH RENAL RESISTIVE INDEX IN A POPULATION-BASED STUDY. J Hypertens, 2015. 33 Suppl 1: p. e15. [9]
  • Ponte, B., et al., Copeptin is associated with kidney length, renal function, and prevalence of simple cysts in a population-based study. J Am Soc Nephrol, 2015. 26(6): p. 1415-25. [10]
  • Pruijm, M., et al., Associations of Urinary Uromodulin with Clinical Characteristics and Markers of Tubular Function in the General Population. Clin J Am Soc Nephrol, 2015. [11]

Under current revision in Psychoneuroendocrinology (22.12.2015)

  • Petrovic, D., et al. Sociodemographic, behavioral and genetic determinants of allostatic load in a Swiss population-based study

2014

  • Alwan, H., et al., Epidemiology of masked and white-coat hypertension: the family-based SKIPOGH study. PLoS One, 2014. 9(3): p. e92522. [12]
  • Ponte, B., et al., Reference values and factors associated with renal resistive index in a family-based population study. Hypertension, 2014. 63(1): p. 136-42. [13]

2013

  • Ehret, G., et al., Genes for blood pressure: an opportunity to understand hypertension. Eur Heart J, 2013. 34(13): p. 951-61. [14]
  • Pruijm, M., et al., Heritability, determinants and reference values of renal length: a family-based population study. Eur Radiol, 2013. 23(10): p. 2899-905. [15]
  • Trudu, M., et al., Common noncoding UMOD gene variants induce salt-sensitive hypertension and kidney damage by increasing uromodulin expression. Nat Med, 2013. 19(12): p. 1655-60. [16]

2012

  • Ehret, G., et al., A multi-SNP locus-association method reveals a substantial fraction of the missing heritability. Am J Hum Genet, 2012. 91(5): p. 863-71. [17]
  • Guessous, I., et al., Caffeine intake and CYP1A2 variants associated with high caffeine intake protect non-smokers from hypertension. Hum Mol Genet, 2012. 21(14): p. 3283-92. [18]

2011

  • Bochud, M., et al., Public health genomics and the challenges for epidemiology. Eur J Public Health, 2011. 21(1): p. 5-6. [19]
  • Ehret, G., et al., Genetic variants in novel pathways influence blood pressure and cardiovascular disease risk. Nature, 2011. 478(7367): p. 103-9. [20]
  • Guessous, I., et al., Calcium, vitamin D and cardiovascular disease. Kidney Blood Press Res, 2011. 34(6): p. 404-17. [21]

2010

  • Ehret, G., Genome-wide association studies: contribution of genomics to understanding blood pressure and essential hypertension. Curr Hypertens Rep, 2010. 12(1): p. 17-25. [22]
  • Ehret, G., et al., Resistant hypertension. Rev Med Suisse, 2010. 6(262): p. 1721-2, 1724-7. [23]
  • Pruijm, M., et al., A new technique for simultaneous validation of two manual nonmercury auscultatory sphygmomanometers (A&D UM-101 and Accoson Greenlight 300) based on the International protocol. Blood Press Monit, 2010. 15(6): p. 322-5. [24]
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