Collaborations and publications

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Caffeine intake and CYP1A2 variants associated with high caffeine intake protect non-smokers from hypertension. Hum Mol Genet. 2012 Jul 15;21(14):3283-92. Guessous I, Dobrinas M, Kutalik Z, Pruijm M, Ehret G, Maillard M, Bergmann S, Beckmann JS, Cusi D, Rizzi F, Cappuccio F, Cornuz J, Paccaud F, Mooser V, Gaspoz JM, Waeber G, Burnier M, Vollenweider P, Eap CB, Bochud M.[http://www.ncbi.nlm.nih.gov/pubmed/22492992]
  
 
A multi-SNP locus-association method reveals a substantial fraction of the missing heritability. Ehret GB, Lamparter D, Hoggart CJ; Genetic Investigation of Anthropometric Traits Consortium, Whittaker JC, Beckmann JS, Kutalik Z.Am J Hum Genet. 2012 Nov 2;91(5):863-71.[http://www.ncbi.nlm.nih.gov/pubmed/23122585]
 
A multi-SNP locus-association method reveals a substantial fraction of the missing heritability. Ehret GB, Lamparter D, Hoggart CJ; Genetic Investigation of Anthropometric Traits Consortium, Whittaker JC, Beckmann JS, Kutalik Z.Am J Hum Genet. 2012 Nov 2;91(5):863-71.[http://www.ncbi.nlm.nih.gov/pubmed/23122585]

Revision as of 20:41, 9 March 2013

2013

Genes for blood pressure: an opportunity to understand hypertension. Ehret GB and Caulfield M. Eur Heart J. 2013[1]


2012

Caffeine intake and CYP1A2 variants associated with high caffeine intake protect non-smokers from hypertension. Hum Mol Genet. 2012 Jul 15;21(14):3283-92. Guessous I, Dobrinas M, Kutalik Z, Pruijm M, Ehret G, Maillard M, Bergmann S, Beckmann JS, Cusi D, Rizzi F, Cappuccio F, Cornuz J, Paccaud F, Mooser V, Gaspoz JM, Waeber G, Burnier M, Vollenweider P, Eap CB, Bochud M.[2]

A multi-SNP locus-association method reveals a substantial fraction of the missing heritability. Ehret GB, Lamparter D, Hoggart CJ; Genetic Investigation of Anthropometric Traits Consortium, Whittaker JC, Beckmann JS, Kutalik Z.Am J Hum Genet. 2012 Nov 2;91(5):863-71.[3]


2011

Bochud M. et al., Eur J Public Health. 2011 Feb;21(1):5-6.[4]

Guessous I et al, Kidney Blood Press Res. 2011 Jun 11;34(6):404-417.[5]

Genetic variants in novel pathways influence blood pressure and cardiovascular disease risk. Ehret GB, Munroe PB, Rice KM, Bochud M et al. Nature. 2011 Sep 11;478(7367):103-9.[6]


2010

Pruijm M et al, Blood Press Monit. 2010 Dec;15(6):322-5.[7]

Resistant Hypertension. Ehret GB, Pechère-Bertschi A. Rev Med Suisse. 2010 Sep 15;6(262):1721-2, 1724-7. [8]

Genome-wide association studies: contribution of genomics to understanding blood pressure and essential hypertension. Ehret GB. Curr Hypertens Rep. 2010 Feb;12(1):17-25.[9]

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